美國(guó)Seracare大腸桿菌O157:H7陽性對(duì)照

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美國(guó)Seracare大腸桿菌O157:H7陽性對(duì)照

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此研究主題刊登于2014年8月的《實(shí)驗(yàn)生物及醫(yī)學(xué)》，證明了丙酮酸脫氫酶復(fù)合體不僅對(duì)于胰島素基因表達(dá)和葡萄糖刺激的胰島素分泌非常必要，同時(shí)還直接影響β-細(xì)胞的生長(zhǎng)和成熟，更闡明了葡萄糖代謝會(huì)直接調(diào)控β細(xì)胞質(zhì)量和可塑性。
紐約州立大學(xué)水牛城分校生物化學(xué)、醫(yī)學(xué)和生物醫(yī)學(xué)科學(xué)學(xué)院的特聘教授，同時(shí)也是資深作者的莫成?帕特爾博士說道：“這些發(fā)現(xiàn)顯示了葡萄糖代謝是調(diào)控β細(xì)胞質(zhì)量的主要調(diào)控因子，很可能是獨(dú)立的訊息傳遞路徑，例如可能不同于胰島素受體作用物2訊息傳遞路徑。”
《實(shí)驗(yàn)生物及醫(yī)學(xué)》期刊主編史蒂芬?古德曼博士說：“帕特等人的研究利用小鼠基因剔除的方式來研究丙酮酸脫氫酶復(fù)合體在胰臟β-細(xì)胞發(fā)育所扮演的角色。他們的研究證明丙酮酸脫氫酶復(fù)合體直接影響β-細(xì)胞的細(xì)胞大小及可塑性。”
盧森堡大學(xué)盧森堡中心系統(tǒng)生物醫(yī)學(xué)研究院(LCSB)的科學(xué)家們已經(jīng)將皮膚細(xì)胞神經(jīng)元重編程植入到小鼠大腦并*維持長(zhǎng)期穩(wěn)定。植入后6個(gè)月，神經(jīng)元功能上*整合到大腦。這一成功，持久穩(wěn)定，神經(jīng)元的植入為將來治療帕金森病人智力時(shí)，用健康的神經(jīng)元替換患病的神經(jīng)元增加希望。
圖片顯示大腦切片部分，其中被移植的誘導(dǎo)神經(jīng)干細(xì)胞*整合到大腦的神經(jīng)元網(wǎng)絡(luò)(藍(lán)色)發(fā)育成一個(gè)復(fù)雜的功能性神經(jīng)元。
LCSB的測(cè)試中，馬克斯普朗克研究所和大學(xué)醫(yī)院明斯特和比勒費(fèi)爾德大學(xué)的研究小組和同事成功將由皮膚細(xì)胞進(jìn)行重編程誘導(dǎo)的神經(jīng)元在小鼠大腦中創(chuàng)建了穩(wěn)定的神經(jīng)組織。產(chǎn)生神經(jīng)元的干細(xì)胞研究者的技術(shù)，或者更具體的說誘導(dǎo)神經(jīng)元干細(xì)胞(iNSC)，在培養(yǎng)皿中培養(yǎng)的宿主皮膚細(xì)胞在很大程度上改善了植入細(xì)胞的相容性。
治療的小鼠沒有顯示出不利的副作用，甚至在植入六個(gè)月后成為大腦的海馬和皮質(zhì)區(qū)域。植入神經(jīng)元*整合到大腦的復(fù)雜網(wǎng)絡(luò)。該神經(jīng)元表現(xiàn)出正?；钚圆⑶彝ㄟ^新形成的突觸——神經(jīng)細(xì)胞之間的接觸點(diǎn)連接到原始腦細(xì)胞。
狡猾的寄生蟲在其生命周期中常常經(jīng)歷顯著的外形變化，讓它們能夠適應(yīng)不同的生活環(huán)境并且繁盛起來。但是發(fā)表在《細(xì)胞生物學(xué)雜志》(The Journal of Cell biology)的一項(xiàng)研究發(fā)現(xiàn)，這些變形看上去可能沒有看上去的那么困難。
非洲的“昏睡病”是一種由稱為布氏錐蟲(Trypanosoma brucei)的由采采蠅傳播的寄生蟲物種導(dǎo)致的疾病。這種單細(xì)胞寄生蟲擁有一個(gè)內(nèi)部?jī)?chǔ)藏著這個(gè)細(xì)胞的線粒體DNA的動(dòng)基體以及對(duì)于細(xì)胞運(yùn)動(dòng)具有關(guān)鍵作用的一個(gè)伸出的鞭毛。布氏錐蟲在其發(fā)育周期經(jīng)歷了外形和構(gòu)成的重大變化。在一個(gè)稱為錐鞭毛體(trypomastigote)的階段，動(dòng)基體位于核之后，而幾乎鞭毛的所有部分都與細(xì)胞相連。另一方面，在短膜蟲期(epimastigote)階段，動(dòng)基體在核之前，而且只有鞭毛的一部分與細(xì)胞相連。布氏錐蟲的近親有許多不同的形狀，這表明這些寄生蟲在進(jìn)化中也改變了它們的形態(tài)。

美國(guó)Seracare

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團(tuán)菌、食品安全、化妝品檢測(cè)、藥物濫用檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

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【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

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The subject of the study, published in August 2014, "Experimental Biology and Medicine," demonstrates that pyruvate dehydrogenase complexes are essential not only for insulin gene expression and glucose-stimulated insulin secretion but also for β-cell growth and Mature, but also shows that glucose metabolism will directly regulate β-cell quality and plasticity.
Dr. Moblie Pa, Distinguished Professor of Biochemistry, Medical and Biomedical Sciences at the State University of New York at Buffalo, said: "These findings show that glucose metabolism is a major regulator of beta-cell mass, Probably an independent messaging pathway, for example, may differ from the insulin receptor 2 signaling pathway. "
Dr. Stephen Goodman, editor-in-chief of Experimental Biology and Medicine, said: "Pat et al's study explored the role of the pyruvate dehydrogenase complex in the pancreatic β-cell development using gene knockout in mice. Their research shows that pyruvate dehydrogenase complex directly affects the cell size and plasticity of β-cells. "
Scientists at the Luxembourg Center for System Biomedicine Research (LCSB) at the University of Luxembourg have already implanted skin-cell neurons into the mouse brains and have maintained long-term stability for the first time. Six months after implantation, neurons functionally integrate compley into the brain. This success, lasting stability, neuronal implantation for the future treatment of Parkinson's patients inligence, with healthy neurons to replace diseased neurons increase hope.
The picture shows a section of the brain that grows into a complex functional neuron in which the transplanted neural stem cells are fully integrated into the brain's neuronal network (blue).
In the latest LCSB test, research groups and colleagues at the Max Planck University and the University Hospital in Münster and Bielefeld succeeded in creating neurons that are reprogrammed by skin cells to create stable Nerve tissue. Stem Cells Producing Neurons Researchers' technology, or more specifically, induction of neuronal stem cells (iNSCs), host skin cells cultured in petri dishes greatly improve the compatibility of the implanted cells.
The treated mice showed no adverse side effects and became brain regions of the hippocampus and cortex even six months after implantation. Implanted neurons are fully integrated into the complex network of the brain. The neurons exhibit normal activity and are connected to the naive brain cells through the contact points between the newly formed synaptic-neurons.
Cunning parasites often experience significant changes in shape throughout their life cycle, allowing them to adapt to different living environments and flourish. But a study published in The Journal of Cell biology found that these distortions may not seem so difficult.
Sleeping sickness in Africa is a disease caused by a parasitic species transmitted by tsetse flies called Trypanosoma brucei. The unicellular parasite has a moving body that holds the mitochondrial DNA of the cell inside, and a protruding flagella that is crucial for cell motility. Trypanosoma brucei undergoes significant changes in shape and composition during its developmental cycle. In a phase called trypomastigote, the motile matrix is ??behind the nucleus, with almost all parts of the flagellum connected to the cell. On the other hand, in the epimastigote stage, the motile matrix precedes the nucleus, and only a portion of the flagella is attached to the cell. The relatives of Trypanosoma brucei have many different shapes, suggesting that these parasites also changed their morphology during evolution.